The Hidden Liquorice: Apparent Mineralocorticoid Excess Caused by Inadvertent Exposure to Liquorice Root Extract
نویسندگان
چکیده
منابع مشابه
APPARENT MINERALOCORTICOID EXCESS IN THREE SIBLINGS
Three siblings (1 boy, 2 girls) with hypertension and hypokalemia are presented, two with low plasma aldosterone and suppressed renin activity and the eldest with a high renin and aldosterone level due to secondary changes in her kidneys. Urinary tetrahydrocortisol (THF) was increased relative to tetrahydrocortisone (THE). Cortisol ring A reduction constant was also lower than normal. Thes...
متن کاملLiquorice: a root cause of secondary hypertension
We describe a patient presenting with hypertension and hypokalaemia who was ultimately diagnosed with liquorice- induced pseudohyperaldosteronism. This rare cause of secondary hypertension illustrates the importance of a methodical approach to the assessment of hypertension.
متن کاملHypertension induced by liquorice tea.
A 45-year-old woman presented to her general practitioner with a 4-month history of hot flushes, sweating and headaches. On examination, she was found to be hypertensive, and blood tests revealed mild hypokalaemia. While awaiting the results of further investigation into the cause of her elevated blood pressure, the patient conducted her own research and identified liquorice tea as the potentia...
متن کاملSyndrome of Apparent Mineralocorticoid Excess
The first adult case of 1 1,-hydroxysteroid dehydrogenase ( 1#6-OHSD) deficiency is described. The impaired conversion of cortisol to cortisone (indicated by urinary cortisol and cortisone metabolites and failure to metabolize 1 a-VIHlcortisol to 13H1H20), was associated with hypertension, hypokalemia, and suppression of the renin-angiotensin-aldosterone system. When established on a fixed Na+/...
متن کاملApparent mineralocorticoid excess (AME) syndrome.
Apparent mineralocorticoid excess (AME) syndrome is a rare autosomal recessive disorder due to the deficiency of 11b hydroxysteroid dehydrogenase type 2 enzyme (11beta-HSD2). Mutations in this gene affect the enzymatic activity resulting to an excess of cortisol, which causes its inappropriate access to mineralocorticoid receptor leading to inherited hypertension.This is a potentially fatal but...
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ژورنال
عنوان ژورنال: AACE Clinical Case Reports
سال: 2015
ISSN: 2376-0605
DOI: 10.4158/ep14556.cr